Dr Charles Hoffe

Dr Charles Hoffe

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Language: English

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00:00
you spoke to a potential mechanism of action of the injury and you mentioned to me the use of a d-dimer test which we all know what it's for but we're gonna have to explain a little bit could you please speak to this a little bit and give the relevant context an introduction please because i think this is really groundbreaking and important yes yeah thank you so so so one of the the key things that really bothered me when i started to see vac serious vaccine injuries in my own
00:32
patients is that i had no idea what the mechanism of injury was and therefore as their doctor i had no idea how to treat it because you know as their family doctor um i i need they would come to me for help and i needed to help them and and i was clueless i mean this was this is an experiment um and i was aware that there was literally um what we call iatrogenic disease a medically induced disease being produced by this vaccine and so so i had asked this in my open
01:05
letter to dr bonnie henry our provincial health officer what is the mechanism mechanism of injury and how do i treat this as these people's doctor and of course nobody knew and and the vaccine manufacturers had told us that the covet spike protein does not go intravenous it stays in the arm the antibodies um to the to the spike protein are produced in the arm um and that's what we had been found but scientists now and dr brody has actually
01:37
very clearly revealed this that only 25 percent of the vaccine actually stays in the arm and the rest of it so these vaccines are are a vast number of little messenger rna strands the moderna vaccine has 40 trillion messenger rna molecules per vaccine dose 40 trillion so so so so these are wrapped in a little lipid capsule the
02:08
little bit capsule is to enable them to be absorbed into the cells so this is injected into the person's arm and their deltoid muscle of the shoulder from there as i mentioned only 25 percent actually stays there the rest is taken up collected through the lymphatic system and fed into the general circulation and so it circulates around the entire body and and i think every doctor knows that absorption from the the circulation occurs in capillary networks because that's where the blood slows right down that it's going through
02:40
tiny tiny vessels so so these little nano capsules containing these trillions of of messenger rna molecules are absorbed into the lining around the capillaries what medically we call the vascular endothelium so so these little cap these little packages are absorbed into the the cells around the vessels the packages open the the body recognizes these messenger rna strands um as as a gene
03:12
and gets to work making curved spike proteins so in a virus those covert spike proteins form part of the viral capsule but the problem is they're not in a virus they're in they're in the cells around blood vessels so as a result they become part of the cell wall of that cell so normally the cells that surround your blood vessels have to be very very smooth to enable
03:41
good and and and unimpeded flow of blood but as soon as you've got all these little spike proteins that become part of the the cell wall it's now a rough surface it's going to be like a very coarse sandpaper it's it's now what the platelets are going to interp interpret as a damaged vessel it's it's no longer smooth it's rough so clotting is inevitable because the platelets that come down that vessel are going to hit a rough spot and assume this must be a damaged vessel this vessel needs to be blocked to stop
04:13
the bleeding that's how our clotting works so so so clots are according because of this and because of the nature of this clots are inevitable because of these um these spike proteins in the capillary networks so i set out to then try and prove this could this theory be correct and and so the problem is these little clots in the capillary networks are microscopic and they are scattered so they're not
04:45
going to show on any scan they're just too small and too scattered it's not like the big clots that cause strokes or heart attacks um they're too small and they're too scattered so how on earth can we know if the person clotted and the only way is with a blood test called a d dimer so the d dimer is a blood test that is that will show up a recent clot it won't show up in old cloth it shows up a new cloth and it doesn't tell you where the clot is it just tells you that the clotting
05:16
mechanism has been activated so i have now been recruiting patients from my practice people that have come into my office and others that have that have heard me speak about this and have asked people to do this d dimer within one week of their covert chart and so far and this study is ongoing these are preliminary results so far i've got 62 percent positive elevated d-dimer which means that the blood clots are not rare that's what the the the the so-called
05:48
experts keep telling us the clots are rare the big ones are rare but the small ones are clearly happening in the majority of people 62 now i'll tell you what the real concern with this is is that a clotted vessel is permanently damaged that vessel never ever goes back to normal so if this theory is correct which it really looks like by these d-dimer results and i'm told it has been done in australia
06:18
and it's been done in the uk and they also found elevated d diamonds um and they they they sort of discarded the information because they said there's no clinical evidence of floods well the clinical the reason is because they're microscopic and they're scattered and so you're not going to see clinical evidence but but in fact all of the frequent side effects of of the of the shock which are headache nausea dizziness fatigue could all be signs of cerebral thrombosis on a
06:48
on a on a capillary level i mean those literally you could be having thousands and thousands of tiny tiny little clots in your brain that won't show on a scan but they will give you those exact symptoms so so the concern is i have now got six people in my medical practice that that cannot exert themselves the way they used to be what what medically we call reduced effort tolerance six people who who now get out of breath doing
07:19
things that they could previously do without any problem so i believe that these people blocked up thousands and thousands of capillaries in their lungs in these six people so i believe these people now have permanently damaged lungs because they have got i mean that's why they get out of breath i have one fellow that used to walk two miles to my office every week for a a shot for his arthritis and he says after a quarter of a mile he's done in other words his effort tolerance is
07:50
reduced to one eighth of what it used to be and and so i've sent some of these people for chest x-rays and ct scans to see what it shows and all it shows is is distorted architecture the the what the radiologist recorded described as increased articulation it's a very non-specific thing and it's because it's microscopic it's just but but but the concern is because these vessels are now permanently damaged in a person's lungs when the heart tries to the heart tries
08:22
to pump blood through all those damaged vessels there's increased resistance trying to pump the blood through those lungs so those people are going to develop something called pulmonary artery hypertension high blood pressure in their lungs and the concern with that is that those people will probably all develop right-sided heart failure within three years and die because they now have increased vascular resistance through their lungs and and lung tissue and heart tissue and
08:53
brain and spinal tissue and all of that does not regenerate in other tissues it can regenerate liver and kidneys and muscle and early but there's some tissues that cannot and and so this is this absolutely explains what i've seen in my patients and that's what i'm doing to prove it and my study is ongoing

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